Transplanted animals were then challenged with a B‐ALL (line #11), which has previously been generated by inducing overexpression of miR‐126 in HSPCs (Nucera et al, 2016) and has extensively been characterized in the context of IFN‐α gene therapy (Escobar et al, 2018). The gene discussed is IFNA1; the disease is acute lymphoblastic leukemia.