AKT1 and gastric cancer: Wound healing, migration, invasion, and colony formation ability of GC cells were greatly increased in the APOC2‐overexpressing group and reduced in the LY294002 (a selective inhibitor of PI3K‐dependent AKT phosphorylation and kinase activity) group, while LY294002 restored the effects of APOC2 overexpression (Figure S8A‐C).