Interestingly, when hBMSCs were treated with overexpressed NEAT1 or PCa‐exosomes, both NEAT1 and NEAT1 shuttled by exosomes (MDA‐PCa‐2b‐NEAT1‐exosomes and C4‐2B‐sh‐NC‐exosomes) were found to significantly promote the RUNX2 expression (Figures 4I,J). Here, RUNX2 is linked to posterior cortical atrophy.