The ALP staining, activity, and mineralization of extracellular matrix of hBMSCs treated with overexpressed NEAT1 (MDA‐PCa‐2b‐NEAT1‐exosomes and C4‐2B‐sh‐NC‐exosomes) were all found to be significantly higher than that of hBMSCs with downregulated NEAT1 (MDA‐PCa‐2b‐NC‐exosomes and C4‐2B‐sh‐NEAT1‐exosomes; Figures 3D–F). Here, NEAT1 is linked to posterior cortical atrophy.