NEAT1 and posterior cortical atrophy: Interestingly, when hBMSCs were treated with overexpressed NEAT1 or PCa‐exosomes, both NEAT1 and NEAT1 shuttled by exosomes (MDA‐PCa‐2b‐NEAT1‐exosomes and C4‐2B‐sh‐NC‐exosomes) were found to significantly promote the RUNX2 expression (Figures 4I,J).