NCOR1 and fatty liver disease: Consistently, the liver‐specific deficiency of CMA precipitates hepatic steatosis (Schneider et al, 2014), and the suppression of hepatic autophagy correlates with defective ketogenesis linked to the accumulation of the autophagy substrate NCOR1 (nuclear receptor co‐repressor 1), which suppresses the PPARA‐dependent transcriptional program of free FA oxidation (Iershov et al, 2019; Saito et al, 2019).