IGF1R and neoplasm: Whole transcriptome analysis of genes and pathways potentially modulating the tumor-initiating capacities and clonogenic functions of DCLK1HI/acetylated α-tubulin (AcTubHI) PDAC cells have revealed upregulation of tuft cell markers (TAS2R31, OR5A2), tubulin acetylation enzyme (ATAT1), Notch response genes (HES1, HES7, and HEY1), proto-oncogene ABL Proto-Oncogene 1 (ABL1), and insulin-like growth factor 1 receptor (IGF-1R) [116].