Furthermore, an experimental mechanism of acute kidney injury has been proposed in both newborn and adult mouse models infected by ZIKV according to a Liu et al. (2019) [40] clinical study on mice, where increasing levels of AKI-related biomarkers (e.g., serum creatinine (Scr), kidney injury molecular−1 (Kim-1), and neutrophil gelatinase-associated lipocalin (NGAL)) were identified [40]. This evidence concerns the gene LCN2 and urogenital neoplasm.