It has been shown that SARS-CoV-2 affects the kidneys and causes podocyte injury that leads to proteinuria and release of proinflammatory cytokines and chemokines, including transforming-growth factor-β (TGF-β), Interferon gamma, interleukins, vascular endothelial growth factors (VEGF), platelet-derived growth factor (PDGF), and chemokine ligand 1 (CXCL1), that causes glomerular sclerosis, hyalinosis, mesangial matrix deposition, and fibrosis [57]. This evidence concerns the gene VEGFA and glomerulosclerosis.