These results (a) support recent reports (20, 24, 25) demonstrating FcγRII/III expression in memory T cells; (b) show that FcγR expression can be further upregulated in disease settings like cancer and viral infections; and (c) demonstrate that a direct link between T cell and B cell immunity as mediated by potential T cell expression of antibody receptors should not be ignored. Here, FCGR2A is linked to viral infectious disease.