We did not detect changes in IFNL2,3, IFNL1, and IFNB1 expression, in agreement with reports of low IFN induction by SARS-CoV-2 infection26,38–41 and the role of several SARS-CoV-2 proteins as IFN-antagonists to limit antiviral response42–44. The gene discussed is IFNL2; the disease is COVID-19.