Furthermore, the potential use of distinct chaperones such as cardiovascular heat shock proteins (HSP), HSPB7, HSPB5 (αBC), HSP70 (HSPA) and HSP90 (HSPC) as dystrophin deficiency disease markers for secondary pathological changes or for the evaluation of novel treatments was also recently proposed [54]. This evidence concerns the gene HSPB7 and neuromuscular disease caused by qualitative or quantitative defects of dystrophin.