MMP2 and pulmonary fibrosis: In this study, the overexpression of THY1 led to a significant decrease in the expression level of Matrix Metallopeptidase 2 (MMP-2), Occludin, α-SMA, Vimentin and β-catenin, and the extent of the β-catenin phosphorylation of pulmonary fibrosis and therefore the inactivation of the WNT signaling pathway.