In line with a previously mentioned study in which PCa patients showed enhanced B2M levels compared to normal [49], we immunocytochemically analyzed MHC class I expression via B2M staining, and the respective cellular state via the common apoptosis marker cleaved Caspase-3 (CASP-3) in primary PCSCs after 24 and 48 h of IFNγ treatment (Figure 6A,B, Figures S7 and S8). Here, CASP3 is linked to posterior cortical atrophy.