Based on the RNA-Seq data from colonic mucosa of C. jejuni-infected patients, activity patterns of vitamin D receptor (VDR)/retinoid X receptor (RXR) pathways were analyzed using Ingenuity Pathway Analysis (IPA) software, which demonstrated that downstream targets related to immune regulation, cell proliferation, and cell differentiation in the patients’ colon mucosae were largely inhibited during campylobacteriosis (Figure 1). Here, VDR is linked to campylobacteriosis.