Given that early AMD is hallmarked by persistent inflammation [35,36], we investigated if FH might influence the levels of relevant inflammatory cytokines, including interleukin-6 (IL6), C-C Motif Chemokine Ligand 2 (CCL2), and interleukin-8 (CXCL8). This evidence concerns the gene CCL2 and familial hyperaldosteronism.