SMARCD3 and dilated cardiomyopathy: One of the few studies regarding the cardiac specific Brg1/Baf60C complex showed that the depletion of Baf60c (mammalian chromatin remodeling complex BRG1-associated factor 60C) in cardiomyocytes results in postnatal dilated cardiomyopathy [34] in a mechanism involving myocardin, a cardiac- and smooth muscle-specific transcription factor involved in cardiac development and homeostasis [62].