BCR and infection: Similar to MPK4, the signaling pathway of Ca2+, acting downstream of CNGCs, was higher expressed at 6 dpi compared to 12 dpi as calcium signaling components including the Calcium-Dependent Protein Kinase (CDPK) and CalModulin (CaM)/CalModulin-Like proteins (CML), supporting that a primary response mechanism was initially activated upon Cmm infection.