In staphylococcal infections, riboflavin exerts an anti-inflammatory effect by reducing NF-κB synthesis, leading to decreased NO and TNFα levels, and modulates the rise of the anti-inflammatory cytokine IL-10 and the function of MCP-1 (monocyte chemoattractant protein 1), a potent chemoattractant [67]. Here, CCL2 is linked to staphylococcal infection.