In view of the finding that the intact Met residues at positions 24, 26, and 27 are related to the ability of CTX1 to induce FasL and Fas expression, it is possible to further use the region around the Met residues of CTX1 to develop peptide mimetics to induce Fas-mediated apoptosis in leukemia cells. The gene discussed is FAS; the disease is leukemia.