The activations of CX3CR1 [93,108,109] and CCR5 [94] chemokine receptors, the CCR2-CCL2 axis stimulation [95] or the overexpression of CCL2, CCL3, CCL4, CCL5, CXCL1 or CX3CL1 on endothelial cells have been reported to contribute to atherosclerosis development via neutrophil recruitment [94,95,107,108,109,110]. Here, CCL2 is linked to atherosclerosis.