Surprisingly, it has been shown that such IFN-γ-activated MSCs did not stimulate allo-reactive T cells but, rather, enhanced their immunosuppressive capacity [13,14], suggesting that MSCs primed with IFN-γ may be used for the treatment of allogeneic conflicts in patients, including graft-versus-host disease [13]. The gene discussed is IFNG; the disease is graft versus host disease.