Interestingly, CCL2, but not CXCL10, was significantly elevated in serum samples of the mdx mouse model for DMD relative to wild-type mice, further suggesting that increased levels of this circulating CCL2 may be associated with muscle pathogenesis due to dystrophin deficiency. The gene discussed is CXCL10; the disease is neuromuscular disease caused by qualitative or quantitative defects of dystrophin.