In the same paper, the authors show that accumulation of NLC1-C in the nucleus promoted proliferation of a testicular embryonal carcinoma cell line by binding to Nucleolin and thus repressing transcription of miR-320a and miR-383, two miRNAs with known oncosuppressive roles that, in turn, negatively regulate NLC1-C expression [71]. The gene discussed is PICSAR; the disease is embryonal carcinoma.