Studies have shown how treatment with 140 μM tea polyphenols induces mitochondrial-mediated apoptosis by activating the caspase-3 and PARP apoptotic cascade in human gallbladder cancer cells [53], 0.5–5 μg/mL of EGCG reduces the cell viability and increases the apoptosis rate of myeloid-derived suppressor cells, mainly through the Arg-1/iNOS/Nox2/NF-κB/STAT3 signaling pathway [54], and 15 μM EGCG inhibits Excision Repair Cross-Complementation Group1/Xeroderma Pigmentosum group F, thereby inhibiting DNA repair and enhancing cisplatin sensitivity in human lung cancer cells [55]. The gene discussed is NFKB1; the disease is lung cancer.