PRDX6 and cataract: Using lens epithelial cells derived from lenses of human, Prdx6-deficient mice, and SCRs (a model for cataract) as model systems, we found a sustained release of encapsulated TAT-HA-Prdx6 analog, and the released TAT-HA-Prdx6 or its mutant at Sumoylation sites was biologically active and retained Prdx6 integrity.