NFKB1 and diffuse large B-cell lymphoma: The genetic aberrations in T-DLBCL, leading to oncogenic signaling, NF-κB pathway activation, and immune-escape phenotype, as well as characteristics of the TME, seem to distinguish T-DLBCL from nodal DLBCL, potentially highlighting the unique pathogenesis of T-DLBCL [12,23,44].