Having shown that the interaction between GSE1 and TACSTD2 plays a critical role in metastatic disease, castration resistance, and disease progression in patients with PCa, we probed the GSE16560/GPL5474 PCa disease progression dataset (n = 281); consistent with previous data, we observed an interesting expression pattern wherein the GSE1 expression ‘peak’ corresponded to a TACSTD2 expression ‘dip’ (Figure 3A). The gene discussed is TACSTD2; the disease is posterior cortical atrophy.