Comparatively in the same study using a rat model of status epilepticus (SE), reduced SV2A immunoreactivity was observed in the inner molecular layer and hilus at 1 day, with increasing loss in the dentate gyrus in the 1 week post-SE, culminating in progressive reductions in SV2A immunoreactivity throughout the hippocampus in the chronic phase at 6 to 8 month post-SE [56]. Here, SV2A is linked to status epilepticus.