From this evidence, researchers have recognized some pathways that are closely related to the pathogenesis of SLE, such as JAK-STAT (STAT1, STAT4) [17], oxidation-related superoxide dismutase (SOD), NADPH [18], apoptosis (Fas, Bcl2, TNF and IFN) [19] and complement and coagulation cascades. This evidence concerns the gene SOAT1 and systemic lupus erythematosus.