AHR and fatty liver disease: For non-EATS modalities, qAOPs concerning immunosuppression and decreased egg production in fish because of GR signaling have been developed (Margiotta-Casaluci et al., 2016), as well as early life stage mortality in birds and fishes via AhR (Doering et al., 2018), and liver steatosis in humans via PPARα (Perkins et al., 2019), among others.