HIF1A and neoplasm: We showed that overexpressed miR-624 led to promoted tumor-forming ability (Figure 5(a)), enlarged tumor volume (Figure 5(b)), increased expression of miR-624 (Figure 5(c)), YAP, HIF1α, VEGF, and Survivin, but reduced ARRDC3 levels (Figure 5(d,e)) as well as stimulated cell proliferation in tumors (Figure 5(f)).