C. trachomatis is an intracellular pathogen that generally triggers a strong host T-helper 1 (Th1) cell and IFN-γ response by the release of chemokines upon infection, and in turn, this could magnify the inflammatory response by recruiting Chlamydia-specific immune cells (Elwell et al., 2016; Vicetti Miguel et al., 2016). Here, IFNG is linked to infection.