KRAS and cervical cancer: These results support our previous finding that inhibition of BCL10 with BCL10 small interfering RNA caused G1 cell cycle arrest in four human cervical cancer cell lines, C33A, HeLa, SiHa, and CaSki, in which all were wild-type KRAS, and silencing BCL10 in SiHa cells significantly downregulated Cyclin D1 expression and the activation of NF-κB [21].