Important findings in this study were (1) the enrichment of a shared (V1−)CEVA haplotype in M1 SLC26A4 cases, (2) two SLC26A4 splice variants and (3) the identification of a FOXI1 variant in three subjects suggesting a contribution of this variant to the etiology of HL and EVA. Here, SLC26A4 is linked to Hodgkins lymphoma.