Nevertheless, as the KLRG‐1+ cells within the lung post‐PbA were primarily IL‐7R–, and as KLRG‐1+IL‐7R– cells have high effector function but low proliferative capacity [55], the impact of these KLRG‐1+ cells compared with traditional Tem cells for tissue protection/pathology following PbA challenge, their durability, and whether they are influenced by repetitive infection, will require further investigation. This evidence concerns the gene KLRG1 and infection.