Though the penetrance was modified by genetic background, the mouse harbouring the Dnah11 gene c.6811G>A (p.Glu2271Lys) variant presents left‐right body patterning asymmetry, static respiratory cilia with normal ultrastructural appearance and reduced sperm motility, accompanied by rhinitis, sinusitis and otitis media, a viable model of human PCD.54, 55. Here, DNAH11 is linked to primary ciliary dyskinesia.