CCL5 and Zika virus infectious disease: As CCL5-KO hBMECs are eliminated by ZIKV infection, and exogenous CCL5 addition restores the viability of ZIKV-infected CCL5-KO cells, our findings support a novel role for ZIKV-elicited CCL5 as a required survival factor that permits ZIKV to persist in hBMECs.