Interestingly, single-nucleotide polymorphisms (SNPs) in LRRK2 have been identified through GWAS as predisposing towards leprosy [117], and a polymorphism in RAB32 [199] has also been associated with disease susceptibility, implicating potential shared pathways in the immune response between LRRK2 and RAB32 and RAB38. Here, RAB38 is linked to leprosy.