PTK2 and neoplasm: Tumor-suppressing properties of RabMab1 were evident in the breast cancer and PDAC settings showing that, when asTF-producing cancer cells were exposed to RabMab1, the activation of FAK, Akt, and MAPK pathways was suppressed in vitro and co-implanting RabMab1 with tumor cells suppressed their growth in vivo (3, 11).