Examining the relationship between MnSOD polymorphisms, a putative risk factor for prostate cancer, and status of exogenous dietary antioxidants—selenium, lycopene, a-tocopherol, and g-tocopherol—in modifying prostate carcinogenesisprocess in 567 cases and 764 controls, Li et al. [297] demonstrated that the combined status of the mentioned antioxidants is inversely associated with the risk of prostate cancer in patients with MnSOD polymorphism. The gene discussed is SOD2; the disease is prostate cancer.