Examining the relationship between MnSOD polymorphisms, a putative risk factor for prostate cancer, and status of exogenous dietary antioxidants—selenium, lycopene, a-tocopherol, and g-tocopherol—in modifying prostate carcinogenesisprocess in 567 cases and 764 controls, Li et al. [297] demonstrated that the combined status of the mentioned antioxidants is inversely associated with the risk of prostate cancer in patients with MnSOD polymorphism. This evidence concerns the gene SOD2 and prostatitis.