Recently, a function of the NUP107–160 complex was disclosed in autoimmunity and spontaneous cell death activated by the simultaneous loss of the receptor-like kinases (RLKs) BRI1-ASSOCIATED KINASE1 (BAK1) and its closest homolog BAK1-LIKE1 (BKK1; Table 3). This evidence concerns the gene BAK1 and Autoimmunity.