The results of our experiments suggested that CGA may display a highly significant anti-inflammatory activity in a well-established mouse model of L-arginine-induced AP as evidenced by reduction of MPO activity and—noteworthy—histopathological changes (H&E staining, Masson’s trichrome staining, IHC/anti-coll-1, IHC/anti-MMP-1). Here, SEMA3A is linked to alkaline phosphatase measurement.