TGFBR1 and Hepatic fibrosis: The miR-942 in activated HSCs was upregulated in cell models and liver specimens of patients with hepatitis B virus-related liver fibrosis and correlated inversely with bone morphogenic proteins and activin membrane-bound inhibitor (BAMBI), which interfered with TGF-β1 signaling by capturing TGF-β receptor I (TβRI/ALK-5) (Tao et al., 2018).