CAMK2G and diabetes mellitus: Although these studies do not directly implicate O-GlcNAc-CaMKII in diabetes, the studied abnormalities are well characterised to be directly affected by CaMKII (Ca2+ fluxand cardiac contraction) so it is not far-fetched to suggest that CaMKII may be playing an intermediary role in these O-GlcNAc mediated dysfunctions.