SUDV (strain Gulu and Boniface)-infected NHPs rapidly become viremic and develop thrombocytopenia, lymphopenia, blood coagulation disorders; increases in liver enzyme activities, including ALT and ALP; and accumulation of serum byproducts of protein metabolism, including BUN and CRE [56,57,58,59,60]. The gene discussed is GPT; the disease is blood coagulation disease.