TNNI3 and COVID-19: Though pathophysiology of cardiovascular manifestations is multifactorial and not yet understood in detail in association with COVID-19 disease, several laboratory findings in COVID-19 patients show an increase in high-sensitivity cardiac troponin I (hs-cTnI), creatine kinase (CK)-myocardial band [31] and N-terminal probrain natriuretic peptide (NT-proBNP), which can be interpreted as an expression of myocardial involvement [246].