When studying AR inhibited tumor growth rate in HepG2 cells resistant to vinblastine, actinomycin D, puromycin, paclitaxel, and doxorubicin, the addition of 25 μg/mL of its 95% ethanol extract significantly lowered the resistance factor of the IC50 ratio of HepG2 MDR subline to HepG2, thus increasing drug sensitivity approximately 3 to 150 times higher than that in HepG2 cells [49]. This evidence concerns the gene AR and neoplasm.