The percentages and numbers of CD8+IFNγ+, CD4+IFNγ+, CD4+IL‐17A+, or γδTCR+IL‐17A+ cells in tumor‐burdened lungs or bronchial draining lymph nodes (dLNs) were comparable between KL and KL9 mice after tumor induction (Figure S4B–F, Supporting Information), indicating that IL‐36γ regulates NSCLC progression independently of lymphocyte differentiation or expansion in vivo. The gene discussed is IL17A; the disease is neoplasm.