IL36G and neoplasm: Knockout, inhibition, or neutralization of IL‐36γ significantly inhibits NSCLC progression and prolongs survival of the KrasLSL‐G12D/+Tp53fl/fl and KrasLSL‐G12D/+Lkb1fl/fl mice after tumor induction, whereas knockout of IL‐36Ra exacerbates tumorigenesis in these NSCLC mouse models and accelerates death of mice.