IL36G and neoplasm: Knockout, inhibition or neutralization of IL‐36γ significantly inhibits NSCLC progression and prolongs survival of the KrasLSL‐G12D/+Tp53fl/fl and KrasLSL‐G12D/+Lkb1fl/fl mice after tumor induction, reduces GSH levels and increases oxidative stress in the tumors and promotes tumor cell death, whereas knockout of IL‐36Ra has opposite effects.