In this context, the percentages or numbers of IL‐17A‐ or IFNγ‐producing CD4+, CD8+, or γδTCR+ cells in the lung tumors were comparable between KL and KL9 or KL and KL5 mice, indicating a dispensable role of IL‐36R signaling in T cell polarization, activation or expansion in the NSCLC mouse model. Here, IFNG is linked to non-small cell lung carcinoma.