However, case reports describing the mineral metabolism profile of patients with familial renal glucosuria, a hereditary condition characterized by inactivating mutation of SGLT2, have reported hypercalciuria, but PTH levels were also found to be in the normal range.(37, 38) Thus, even in the context of glucosuria‐induced hypercalciuria in the setting of SGLT2 inhibition, hypercalciuria may not be marked enough to cause detectable alterations in calciotropic hormones. This evidence concerns the gene PTH and familial renal glucosuria.