Transforming growth factor-β signaling, which was reported to be upregulated in muscle samples with sporadic inclusion body myositis [41], and mTOR signaling may be potential pathways of autoimmune diseases [42] and were included in the CTGs of DE miRNAs in the DM-nonILD-MSA16(-) exosomes, suggesting that these pathways may contribute to the pathogenesis of DM. The gene discussed is MTOR; the disease is dermatomyositis.