Some of these mechanisms reported with the use of B. subtilis are: 1) sustained increase in mucin secretion (Jacquier et al., 2019) (which plays a vital role in maintaining the architecture of the mucus layer on the intestinal surface); 2) significant increase in goblet cells (Camargo et al., 2012) (which directly affects the innate immune response and regulates the response to inflammation/infection); and 3) increase in the reactions of the mucous membranes to pathogens and putrefaction agents (Barrera et al., 2014). This evidence concerns the gene MUC5AC and infection.